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Table 5 Results of multivariable regression models for PR layer thickness in the SLE group

From: Neurodegeneration in systemic lupus erythematosus: layer by layer retinal study using optical coherence tomography

Model*Coefficient estimate95% confidence intervalp-value
Dependent variable: PR layer thickness at C sector
 NP-SLE− 2.83− 5.29 to − 0.370.025
 Sjogren’s syndrome5.050.71 to 9.400.023
 SLEDAI− 0.29− 0.51 to − 0.060.015
 Body mass index (kg/m2)− 0.31− 0.60 to − 0.020.038
Dependent variable: PR layer thickness at S3 sector
 Benzodiazepine2.670.35 to 4.990.025
Dependent variable: PR layer thickness at T3 sector
 Calcium channel blocker− 2.68− 5.23 to − 0.140.039
Dependent variable: PR layer thickness at I3 sector
 Statin− 1.84− 3.48 to − 0.200.028
Dependent variable: PR layer thickness at N3 sector
 Sjogren’s syndrome2.380.2 to 4.740.048
 SLEDAI− 0.13− 0.26 to 0.000.050
 Statin− 2.44− 4.15 to − 0.730.006
Dependent variable: PR layer thickness at S6 sector
 Benzodiazepine1.950.23 to 3.680.027
 HCQ treatment time (years)− 0.83− 0.16 to 0.000.041
Dependent variable: PR layer thickness at N6 sector
 Axial length (mm)0.530.01 to 1.050.045
 ACE inhibitor− 1.50− 2.95 to − 0.050.043
  1. No multiple model was achieved for the T6 and I6 sectors. p-values were obtained by linear regression models
  2. ACE angiotensin-converting enzyme, HCQ hydroxychloroquine, NP-SLE neuropsychiatric systemic lupus erythematosus, PR photoreceptor, SLEDAI Systemic Lupus Erythematosus Disease Activity Index